bulbar ataxia - vertaling naar arabisch
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bulbar ataxia - vertaling naar arabisch

HUMAN DISEASE
Friedreich ataxia; Friedrich's ataxia; Friedreich's Ataxis; Friedreich's Ataxia; Friedreich’s ataxia; Friedreichs Ataxia; Friedrich ataxia; Freidrich's ataxia; Freidreich's ataxia; Friedrick's ataxia; Freidrick's ataxia; Frederick's ataxia; Frederich's ataxia; Friederich's ataxia; Friederick's ataxia; Freiderick's ataxia; Freiderich's ataxia; Fredrick's ataxia; Fredrich's ataxia; FRDA
  • alt=Photo of Kyle Bryant training on his recumbent bicycle
  • alt=Photo of Nikolaus Friedreich

bulbar ataxia      
‎ رَنَحٌ بَصَلِيُّ المَنْشَأ‎
ataxia telangiectasia syndrome         
  • Ocular telangiectasia in a person with A–T
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  • Comparison of clinical and laboratory features of rare genetic disorders than can be confused with A–T
A RARE, NEURODEGENERATIVE, AUTOSOMAL RECESSIVE HUMAN DISEASE CAUSING SEVERE DISABILITY
Ataxia Telangiectasia; A-T; Boder-Sedgwick syndrome; Louis Bar syndrome; Louis-Bar syndrome; Ataxia telangectasia; Louis–Bar syndrome; Ataxia telangiectasia syndrome; Ataxia telangiectasia; Ataxia-telangiectasia
‎ مُتَلاَزِمَةُ الرَّنَح و تَوَسُّعُ الشُّعَيرات‎
progressive bulbar paralysis         
HUMAN DISEASE
Bulbar palsy, progressive; Progressive bulbar paralysis; Progressive Bulbar Palsy
‎ الشَّلَلُ البَصَلِيُّ المُتَرَقِّي‎

Definitie

ataxia
[?'taks??]
(also ataxy -si)
¦ noun Medicine the loss of full control of bodily movements.
Derivatives
ataxic adjective
Origin
C19: mod. L., from Gk, from a- 'without' + taxis 'order'.

Wikipedia

Friedreich's ataxia

Friedreich's ataxia (FRDA or FA) is an autosomal-recessive genetic disease that causes difficulty walking, a loss of sensation in the arms and legs, and impaired speech that worsens over time. Symptoms generally start between 5 and 20 years of age. Many develop hypertrophic cardiomyopathy and require a mobility aid such as a cane, walker, or wheelchair in their teens. As the disease progresses, some affected people lose their sight and hearing. Other complications may include scoliosis and diabetes mellitus.

The condition is caused by mutations in the FXN gene on chromosome 9, which makes a protein called frataxin. In FRDA, cells produce less frataxin. Degeneration of nerve tissue in the spinal cord causes the ataxia; particularly affected are the sensory neurons essential for directing muscle movement of the arms and legs through connections with the cerebellum. The spinal cord becomes thinner, and nerve cells lose some myelin sheath.

No effective treatment is known, but several therapies are in trials. FRDA shortens life expectancy due to heart disease, but some people can live into their 60s or older.

FRDA affects one in 50,000 people in the United States and is the most common inherited ataxia. Rates are highest in people of Western European descent. The condition is named after German physician Nikolaus Friedreich, who first described it in the 1860s.