gelatin coagulation - определение. Что такое gelatin coagulation
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Что (кто) такое gelatin coagulation - определение

PHOTOGRAPHIC PROCESS
Gelatin silver print; Gelatin silver; Gelatin-silver print; Gelatin-silver; Gelatin Process; Dry plate; Gelatin process; Dry plate photography; Dry Plate; Gelatin-silver process; Silver gelatin; Gelatin dry plate negative; Gelatin dry glass plate negative; Gelatin developing out paper (DOP)
  • Childe Hassam by James W. Porter, 1913, silver print
  • A gelatin silver print of a Hawaiian girl
Найдено результатов: 104
Gelatin silver process         
The gelatin silver process is the most commonly used chemical process in black-and-white photography, and is the fundamental chemical process for modern analog color photography. As such, films and printing papers available for analog photography rarely rely on any other chemical process to record an image.
Dry plate         
Dry plate, also known as gelatin process, is an improved type of photographic plate. It was invented by Dr.
Coagulopathy         
CONDITION IN WHICH THE BLOOD’S ABILITY TO COAGULATE (FORM CLOTS) IS IMPAIRED
Blood coagulation disorders; Coagulation protein disorders; Bleeding disorder; Bleeding disorders; Coagulation disturbance; Coagulation defects; Coagulation disorder; Blood Coagulation Disorders, Inherited; Inherited Blood Coagulation Disorders; Blood clotting disorder; Bruising/purpura due to coagulation disorder; Blood clotting disorders; Clotting disorder; Trauma-induced coagulopathy; Blood clotting problems; Clotting disorders; Dilutional coagulopathy; Bleeding problems; Hemostatic disorder; Disorders of coagulation
Coagulopathy (also called a bleeding disorder) is a condition in which the blood's ability to coagulate (form clots) is impaired. This condition can cause a tendency toward prolonged or excessive bleeding (bleeding diathesis), which may occur spontaneously or following an injury or medical and dental procedures.
coagulate         
  • isbn=978-1-904842-39-2}}</ref>
  • Coagulation with arrows for negative and positive feedback.
  • enzymatic activity]].
  • GPV]] allows for platelets to adhere to the site of injury. Mutations in the genes associated with the glycoprotein Ib-IX-V complex are characteristic of [[Bernard–Soulier syndrome]]
  • Modern coagulation pathway. Hand-drawn composite from similar drawings presented by Professor Dzung Le, MD, PhD, at UCSD Clinical Chemistry conferences on 14 and 21 October 2014. Original schema from Introduction to Hematology by Samuel I. Rapaport. 2nd edition;Lippencott:1987. Dr Le added the factor XI portion based on a paper from about year 2000. Dr. Le's similar drawings presented the development of this cascade over 6 frames, like a comic.
  • The interaction of vWF and GP1b alpha. The GP1b receptor on the surface of platelets allows the platelet to bind to vWF, which is exposed upon damage to vasculature. The vWF A1 domain (yellow) interacts with the extracellular domain of GP1ba (blue).
PROCESS IN WHICH THE COAGULATION FACTORS OF THE BLOOD INTERACT, ULTIMATELY RESULTING A CLOT
Blood coagulation; Coagulation factors; Coagulation factor; Blood clotting; Clotting factors; Clotting factor; Common pathway; Clotting; Coagulation system; Coagulate; Blood Clotting; Coagulation of human blood; Coagulation cascade; Blood clotting spray; Blood Clotting Spray; Clotting cascade; Coagulating; Coagulative; Blood coagulation factors; Blood coagulation tests; Blood coagulation factor inhibitors; Blood clotting cascade; Grumous; Secondary hemostasis; Blood-clotting; Blood clotting mechanism; Clot formation; Factor 4; Factor IV; Clotting system; Procoagulant; Platelet activation; Thrombokinesis; Primary hemostasis; Form clots; Tissue factor pathway; Blood clotting factors; Blood clotting factor; Coagulation protein; Coagulation proteins; Bloodclotting; Procoagulation; Proagulatory; Procoagulatory; Pro-coagulatory; Pro-coagulant; Pro-coagulation; Coagulation pathway; Fibrinogenesis; Coagulation activation; Fibrin generation; Fibrin formation; Fibrin genesis; Fibrin production; Fibrin synthesis
(coagulates, coagulating, coagulated)
When a liquid coagulates, it becomes very thick.
The blood coagulates to stop wounds bleeding.
= congeal
VERB: V
coagulation
Blood becomes stickier to help coagulation in case of a cut.
N-UNCOUNT
Coagulating         
  • isbn=978-1-904842-39-2}}</ref>
  • Coagulation with arrows for negative and positive feedback.
  • enzymatic activity]].
  • GPV]] allows for platelets to adhere to the site of injury. Mutations in the genes associated with the glycoprotein Ib-IX-V complex are characteristic of [[Bernard–Soulier syndrome]]
  • Modern coagulation pathway. Hand-drawn composite from similar drawings presented by Professor Dzung Le, MD, PhD, at UCSD Clinical Chemistry conferences on 14 and 21 October 2014. Original schema from Introduction to Hematology by Samuel I. Rapaport. 2nd edition;Lippencott:1987. Dr Le added the factor XI portion based on a paper from about year 2000. Dr. Le's similar drawings presented the development of this cascade over 6 frames, like a comic.
  • The interaction of vWF and GP1b alpha. The GP1b receptor on the surface of platelets allows the platelet to bind to vWF, which is exposed upon damage to vasculature. The vWF A1 domain (yellow) interacts with the extracellular domain of GP1ba (blue).
PROCESS IN WHICH THE COAGULATION FACTORS OF THE BLOOD INTERACT, ULTIMATELY RESULTING A CLOT
Blood coagulation; Coagulation factors; Coagulation factor; Blood clotting; Clotting factors; Clotting factor; Common pathway; Clotting; Coagulation system; Coagulate; Blood Clotting; Coagulation of human blood; Coagulation cascade; Blood clotting spray; Blood Clotting Spray; Clotting cascade; Coagulating; Coagulative; Blood coagulation factors; Blood coagulation tests; Blood coagulation factor inhibitors; Blood clotting cascade; Grumous; Secondary hemostasis; Blood-clotting; Blood clotting mechanism; Clot formation; Factor 4; Factor IV; Clotting system; Procoagulant; Platelet activation; Thrombokinesis; Primary hemostasis; Form clots; Tissue factor pathway; Blood clotting factors; Blood clotting factor; Coagulation protein; Coagulation proteins; Bloodclotting; Procoagulation; Proagulatory; Procoagulatory; Pro-coagulatory; Pro-coagulant; Pro-coagulation; Coagulation pathway; Fibrinogenesis; Coagulation activation; Fibrin generation; Fibrin formation; Fibrin genesis; Fibrin production; Fibrin synthesis
·p.pr. & ·vb.n. of Coagulate.
Coagulation         
  • isbn=978-1-904842-39-2}}</ref>
  • Coagulation with arrows for negative and positive feedback.
  • enzymatic activity]].
  • GPV]] allows for platelets to adhere to the site of injury. Mutations in the genes associated with the glycoprotein Ib-IX-V complex are characteristic of [[Bernard–Soulier syndrome]]
  • Modern coagulation pathway. Hand-drawn composite from similar drawings presented by Professor Dzung Le, MD, PhD, at UCSD Clinical Chemistry conferences on 14 and 21 October 2014. Original schema from Introduction to Hematology by Samuel I. Rapaport. 2nd edition;Lippencott:1987. Dr Le added the factor XI portion based on a paper from about year 2000. Dr. Le's similar drawings presented the development of this cascade over 6 frames, like a comic.
  • The interaction of vWF and GP1b alpha. The GP1b receptor on the surface of platelets allows the platelet to bind to vWF, which is exposed upon damage to vasculature. The vWF A1 domain (yellow) interacts with the extracellular domain of GP1ba (blue).
PROCESS IN WHICH THE COAGULATION FACTORS OF THE BLOOD INTERACT, ULTIMATELY RESULTING A CLOT
Blood coagulation; Coagulation factors; Coagulation factor; Blood clotting; Clotting factors; Clotting factor; Common pathway; Clotting; Coagulation system; Coagulate; Blood Clotting; Coagulation of human blood; Coagulation cascade; Blood clotting spray; Blood Clotting Spray; Clotting cascade; Coagulating; Coagulative; Blood coagulation factors; Blood coagulation tests; Blood coagulation factor inhibitors; Blood clotting cascade; Grumous; Secondary hemostasis; Blood-clotting; Blood clotting mechanism; Clot formation; Factor 4; Factor IV; Clotting system; Procoagulant; Platelet activation; Thrombokinesis; Primary hemostasis; Form clots; Tissue factor pathway; Blood clotting factors; Blood clotting factor; Coagulation protein; Coagulation proteins; Bloodclotting; Procoagulation; Proagulatory; Procoagulatory; Pro-coagulatory; Pro-coagulant; Pro-coagulation; Coagulation pathway; Fibrinogenesis; Coagulation activation; Fibrin generation; Fibrin formation; Fibrin genesis; Fibrin production; Fibrin synthesis
Coagulation, also known as clotting, is the process by which blood changes from a liquid to a gel, forming a blood clot. It potentially results in hemostasis, the cessation of blood loss from a damaged vessel, followed by repair.
Coagulate         
  • isbn=978-1-904842-39-2}}</ref>
  • Coagulation with arrows for negative and positive feedback.
  • enzymatic activity]].
  • GPV]] allows for platelets to adhere to the site of injury. Mutations in the genes associated with the glycoprotein Ib-IX-V complex are characteristic of [[Bernard–Soulier syndrome]]
  • Modern coagulation pathway. Hand-drawn composite from similar drawings presented by Professor Dzung Le, MD, PhD, at UCSD Clinical Chemistry conferences on 14 and 21 October 2014. Original schema from Introduction to Hematology by Samuel I. Rapaport. 2nd edition;Lippencott:1987. Dr Le added the factor XI portion based on a paper from about year 2000. Dr. Le's similar drawings presented the development of this cascade over 6 frames, like a comic.
  • The interaction of vWF and GP1b alpha. The GP1b receptor on the surface of platelets allows the platelet to bind to vWF, which is exposed upon damage to vasculature. The vWF A1 domain (yellow) interacts with the extracellular domain of GP1ba (blue).
PROCESS IN WHICH THE COAGULATION FACTORS OF THE BLOOD INTERACT, ULTIMATELY RESULTING A CLOT
Blood coagulation; Coagulation factors; Coagulation factor; Blood clotting; Clotting factors; Clotting factor; Common pathway; Clotting; Coagulation system; Coagulate; Blood Clotting; Coagulation of human blood; Coagulation cascade; Blood clotting spray; Blood Clotting Spray; Clotting cascade; Coagulating; Coagulative; Blood coagulation factors; Blood coagulation tests; Blood coagulation factor inhibitors; Blood clotting cascade; Grumous; Secondary hemostasis; Blood-clotting; Blood clotting mechanism; Clot formation; Factor 4; Factor IV; Clotting system; Procoagulant; Platelet activation; Thrombokinesis; Primary hemostasis; Form clots; Tissue factor pathway; Blood clotting factors; Blood clotting factor; Coagulation protein; Coagulation proteins; Bloodclotting; Procoagulation; Proagulatory; Procoagulatory; Pro-coagulatory; Pro-coagulant; Pro-coagulation; Coagulation pathway; Fibrinogenesis; Coagulation activation; Fibrin generation; Fibrin formation; Fibrin genesis; Fibrin production; Fibrin synthesis
·vi To undergo coagulation.
II. Coagulate ·adj Coagulated.
III. Coagulate ·vt To cause (a liquid) to change into a curdlike or semisolid state, not by evaporation but by some kind of chemical reaction; to Curdle; as, rennet coagulates milk; heat coagulates the white of an Egg.
Coagulative         
  • isbn=978-1-904842-39-2}}</ref>
  • Coagulation with arrows for negative and positive feedback.
  • enzymatic activity]].
  • GPV]] allows for platelets to adhere to the site of injury. Mutations in the genes associated with the glycoprotein Ib-IX-V complex are characteristic of [[Bernard–Soulier syndrome]]
  • Modern coagulation pathway. Hand-drawn composite from similar drawings presented by Professor Dzung Le, MD, PhD, at UCSD Clinical Chemistry conferences on 14 and 21 October 2014. Original schema from Introduction to Hematology by Samuel I. Rapaport. 2nd edition;Lippencott:1987. Dr Le added the factor XI portion based on a paper from about year 2000. Dr. Le's similar drawings presented the development of this cascade over 6 frames, like a comic.
  • The interaction of vWF and GP1b alpha. The GP1b receptor on the surface of platelets allows the platelet to bind to vWF, which is exposed upon damage to vasculature. The vWF A1 domain (yellow) interacts with the extracellular domain of GP1ba (blue).
PROCESS IN WHICH THE COAGULATION FACTORS OF THE BLOOD INTERACT, ULTIMATELY RESULTING A CLOT
Blood coagulation; Coagulation factors; Coagulation factor; Blood clotting; Clotting factors; Clotting factor; Common pathway; Clotting; Coagulation system; Coagulate; Blood Clotting; Coagulation of human blood; Coagulation cascade; Blood clotting spray; Blood Clotting Spray; Clotting cascade; Coagulating; Coagulative; Blood coagulation factors; Blood coagulation tests; Blood coagulation factor inhibitors; Blood clotting cascade; Grumous; Secondary hemostasis; Blood-clotting; Blood clotting mechanism; Clot formation; Factor 4; Factor IV; Clotting system; Procoagulant; Platelet activation; Thrombokinesis; Primary hemostasis; Form clots; Tissue factor pathway; Blood clotting factors; Blood clotting factor; Coagulation protein; Coagulation proteins; Bloodclotting; Procoagulation; Proagulatory; Procoagulatory; Pro-coagulatory; Pro-coagulant; Pro-coagulation; Coagulation pathway; Fibrinogenesis; Coagulation activation; Fibrin generation; Fibrin formation; Fibrin genesis; Fibrin production; Fibrin synthesis
·adj Having the power to cause coagulation; as, a coagulative agent.
grumous         
  • isbn=978-1-904842-39-2}}</ref>
  • Coagulation with arrows for negative and positive feedback.
  • enzymatic activity]].
  • GPV]] allows for platelets to adhere to the site of injury. Mutations in the genes associated with the glycoprotein Ib-IX-V complex are characteristic of [[Bernard–Soulier syndrome]]
  • Modern coagulation pathway. Hand-drawn composite from similar drawings presented by Professor Dzung Le, MD, PhD, at UCSD Clinical Chemistry conferences on 14 and 21 October 2014. Original schema from Introduction to Hematology by Samuel I. Rapaport. 2nd edition;Lippencott:1987. Dr Le added the factor XI portion based on a paper from about year 2000. Dr. Le's similar drawings presented the development of this cascade over 6 frames, like a comic.
  • The interaction of vWF and GP1b alpha. The GP1b receptor on the surface of platelets allows the platelet to bind to vWF, which is exposed upon damage to vasculature. The vWF A1 domain (yellow) interacts with the extracellular domain of GP1ba (blue).
PROCESS IN WHICH THE COAGULATION FACTORS OF THE BLOOD INTERACT, ULTIMATELY RESULTING A CLOT
Blood coagulation; Coagulation factors; Coagulation factor; Blood clotting; Clotting factors; Clotting factor; Common pathway; Clotting; Coagulation system; Coagulate; Blood Clotting; Coagulation of human blood; Coagulation cascade; Blood clotting spray; Blood Clotting Spray; Clotting cascade; Coagulating; Coagulative; Blood coagulation factors; Blood coagulation tests; Blood coagulation factor inhibitors; Blood clotting cascade; Grumous; Secondary hemostasis; Blood-clotting; Blood clotting mechanism; Clot formation; Factor 4; Factor IV; Clotting system; Procoagulant; Platelet activation; Thrombokinesis; Primary hemostasis; Form clots; Tissue factor pathway; Blood clotting factors; Blood clotting factor; Coagulation protein; Coagulation proteins; Bloodclotting; Procoagulation; Proagulatory; Procoagulatory; Pro-coagulatory; Pro-coagulant; Pro-coagulation; Coagulation pathway; Fibrinogenesis; Coagulation activation; Fibrin generation; Fibrin formation; Fibrin genesis; Fibrin production; Fibrin synthesis
a.
Clotted, concreted, thick.
coagulation         
  • isbn=978-1-904842-39-2}}</ref>
  • Coagulation with arrows for negative and positive feedback.
  • enzymatic activity]].
  • GPV]] allows for platelets to adhere to the site of injury. Mutations in the genes associated with the glycoprotein Ib-IX-V complex are characteristic of [[Bernard–Soulier syndrome]]
  • Modern coagulation pathway. Hand-drawn composite from similar drawings presented by Professor Dzung Le, MD, PhD, at UCSD Clinical Chemistry conferences on 14 and 21 October 2014. Original schema from Introduction to Hematology by Samuel I. Rapaport. 2nd edition;Lippencott:1987. Dr Le added the factor XI portion based on a paper from about year 2000. Dr. Le's similar drawings presented the development of this cascade over 6 frames, like a comic.
  • The interaction of vWF and GP1b alpha. The GP1b receptor on the surface of platelets allows the platelet to bind to vWF, which is exposed upon damage to vasculature. The vWF A1 domain (yellow) interacts with the extracellular domain of GP1ba (blue).
PROCESS IN WHICH THE COAGULATION FACTORS OF THE BLOOD INTERACT, ULTIMATELY RESULTING A CLOT
Blood coagulation; Coagulation factors; Coagulation factor; Blood clotting; Clotting factors; Clotting factor; Common pathway; Clotting; Coagulation system; Coagulate; Blood Clotting; Coagulation of human blood; Coagulation cascade; Blood clotting spray; Blood Clotting Spray; Clotting cascade; Coagulating; Coagulative; Blood coagulation factors; Blood coagulation tests; Blood coagulation factor inhibitors; Blood clotting cascade; Grumous; Secondary hemostasis; Blood-clotting; Blood clotting mechanism; Clot formation; Factor 4; Factor IV; Clotting system; Procoagulant; Platelet activation; Thrombokinesis; Primary hemostasis; Form clots; Tissue factor pathway; Blood clotting factors; Blood clotting factor; Coagulation protein; Coagulation proteins; Bloodclotting; Procoagulation; Proagulatory; Procoagulatory; Pro-coagulatory; Pro-coagulant; Pro-coagulation; Coagulation pathway; Fibrinogenesis; Coagulation activation; Fibrin generation; Fibrin formation; Fibrin genesis; Fibrin production; Fibrin synthesis

Википедия

Gelatin silver process

The gelatin silver process is the most commonly used chemical process in black-and-white photography, and is the fundamental chemical process for modern analog color photography. As such, films and printing papers available for analog photography rarely rely on any other chemical process to record an image. A suspension of silver salts in gelatin is coated onto a support such as glass, flexible plastic or film, baryta paper, or resin-coated paper. These light-sensitive materials are stable under normal keeping conditions and are able to be exposed and processed even many years after their manufacture. The "dry plate" gelatin process was an improvement on the collodion wet-plate process dominant from the 1850s–1880s, which had to be exposed and developed immediately after coating.